Alcohol causes different cancers

The more alcohol you drink, the higher your risk of developing at least seven different types of cancers. Drinking small or moderate amounts of alcohol was associated with increased risks for esophogeal, mouth, voice box, liver, stomach, pancreas and breast cancers, and is responsible for more than five percent of cancers and cancer deaths worldwide.

The American Society of Clinical Oncology ASCO has never addressed the link between alcohol and cancer, but is now underscoring the importance of controlling the risk of alcohol consumption to reducing the risk of cancer. While the ASCO does suggest strategies for cutting back on drinking, it also advocates for temperate use of alcohol, rather than recommending give up drinking.

The Centres for Disease Control and Prevention CDC recommends that women should not have more than one drink a day or eight drinks a week. Men drink two drinks a day, or 14 a week. There has been some debate over whether alcohol or other compositions of various alcoholic beverages are cancer-causing. There is an associations between alcohol drinking and cancer risk. Alcohol does not affect each part of the body in the same carcinogenic way.

For head and neck and esophageal cancers, alcohol's breakdown product- acetaldehyde, which is an established carcinogen touches the tissues directly as drinker swallows an alcoholic drink and causes cancer. Liver cancer is caused by cirrhosis, which is caused by drinking. When cirrhosis develops, healthy liver cells are replaced by damaged scar tissue cells, which can become cancer cells. It interferes with the absorption of folate, which leads to development of colon cancer.

When a woman’s estrogen levels become abnormally high, the hormone puts her at higher risk for breast cancer. Alcohol has been shown to increase estrogen levels, thus putting women at greater risk of breast cancer. In fact, ASCO reports that women who drank even one drink of beer or wine which have significantly lower alcohol contents than liquors were five percent more likely to develop premenopausal breast cancer, and nine percent more likely to develop the cancer after menopause.
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Links between cancerous toxin and cannabis extract

Researchers discovered benzene and other potentially cancer-causing chemicals in the vapor produced by butane hash oil, a cannabis extract.

This raises health concerns about dabbing, or vaporizing hash oil - a practice that is growing in popularity, especially in states that have legalized medical or recreational marijuana.

Dabbing is placing of a small amount of cannabis extract-a dab on a heated surface and inhaling the produced vapor. The practice has raised concerns because it produces extremely high levels of cannabinoids -the active ingredients in marijuana.

The process of making hash oil also is dangerous because it uses highly flammable and potentially explosive butane as a solvent to extract active ingredients from marijuana leaves and flowers. Dabbinge- a form of vaporization, may produce high amounts of toxins, researchers analyzed the chemical profile of terpenes - the fragrant oils in marijuana and other plants - by vaporizing them in much the same way as a user would vaporize hash oil.

Terpenes are used in e-cigarette liquids, toxic chemicals in e-cigarette vapor when the devices were used at high temperature is very dangerous. 

The dabbing experiments produced benzene - a carcinogen at levels at higher level than the ambient air, It also produced high levels of methacrolein, a chemical similar to acrolein, also a carcinogen.
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E-cigarette with nicotine changes adrenaline in nonsmokers heart

Healthy nonsmokers experienced increased adrenaline levels in their heart after taking one electronic cigarette with nicotine but there were no increased adrenaline levels when the study subjects used a nicotine-free or empty e-cigarette.

Unlike cigarettes, e-cigarette have no combustion or tobacco. Instead, these electronic, handheld devices deliver nicotine with flavoring and other chemicals in a vapor instead of smoke.
E-cigarettes produce fewer carcinogens than tar of tobacco cigarette smoke, they also produce nicotine.

E-cigarette users have elevated sympathetic nerve activity which increases adrenaline directed to the heart and are more susceptible to oxidative stress. Researchers used heart rate variability obtained from a prolonged, non-invasive heart rhythm recording. Heart rate variability is calculated from the degree of variability in the time between heartbeats.

This variability may be indicative of the amount of adrenaline on the heart.
heart rate variability test to link increased adrenaline activity in the heart with increased cardiac risk. People with known heart disease and people without known heart disease who have this pattern of high adrenaline levels in the heart have increased risk of death.

In the first study to separate the nicotine from the non-nicotine components when looking at the heart impact of e-cigarettes on humans, researchers studied healthy adults who were not smoking. Researchers measured cardiac adrenaline activity by assessing heart rate variability and oxidative stress in blood samples by measuring the enzyme plasma paraoxonase PON1.

They discovered that exposure to e-cigarettes with nicotine, but not e-cigarettes without nicotine, led to increased adrenaline levels to the heart, as indicated by abnormal heart rate variability. Acute electronic cigarette use with nicotine increases cardiac adrenaline levels. And it's in the same pattern that is associated with increased cardiac risk in patients who have known cardiac disease and even in patients without known cardiac disease.
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